Astragalus is a plant native to Asia, considered to be one of the most important herbs in Traditional Chinese Medicine. It is used for the common cold, upper respiratory infections, allergies, fibromyalgia, anemia, HIV/AIDS and to strengthen and regulate the immune system. It also relieves chronic fatigue syndrome (CFS), kidney disease, diabetes, and high blood pressure. Astragalus can be used as a general tonic, to protect the liver and to fight bacteria and viruses. It is often combined with other herbs, for example, in combination with ligustrum lucidum (glossy privet), astragalus is used orally for treating breast cancer, cervical cancer, and lung cancer.
Cycloastragenol powder has a similar chemical structure to that of the Astragaloside IV molecule, but it is smaller and significantly more bioavailable, enabling lower doses to be taken. It is already used as an immunostimulant because of its ability to increase T lymphocyte proliferation. However, it is its exceptional anti-aging properties that are of increasing interest to the scientific community.
| Product Name |
Cycloastragenol |
| Latin Name |
Astragalus Membranaceus (Fisch.) Bunge |
| Specification |
5% to 99% |
| Appearance: |
White powder |
| Molecular Formula |
C30H50O5 |
| Molecular Weight |
490.72 |
| Extraction Type |
Ethanol extraction |
| Test Method |
HPLC-ELSD |
Cycloastragenol has a similar chemical structure to that of the Astragaloside IV molecule, but it is smaller and significantly more bioavailable, enabling lower doses to be taken. It is already used as an immunostimulant because of its ability to increase T lymphocyte proliferation. However, it is its exceptional anti-aging properties that are of increasing interest to the scientific community.
Cycloastragenol stimulates the repair of DNA damage by activating telomerase, a nucleoprotein enzyme which catalyses synthesis and growth of telomeric DNA. Telomeres are made of thin filaments and are found at the tips of chromosomes. Maintaining their stability enables cells to avoid replicative senescence and indefinite proliferation beyond the 'Hayflick limit'. Telomeres shorten with each cycle of cell division, or when subjected to oxidative stress. Until now, this has been an unavoidable mechanism of aging.
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